Simini Boards Cast
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🧠 Flashcard-style recaps + board-style questions
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Simini Boards Cast
Chapter 112 - Part A: The Growth Program: Why the Prostate Never Stops Enlarging
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In this BoardsCast episode, we begin Tobias Chapter 113 — Prostate with the reframe that makes BPH predictable instead of “mysterious”:
The prostate isn’t malfunctioning. It’s following the program.
From puberty onward, the canine prostate stays under continuous androgen stimulation. The gland is the only accessory sex gland in the male dog, it encircles the proximal urethra, and it keeps growing year after year because the signal never stops.
We build the core cascade you must know cold:
Androgen signal → DHT activation → tissue accumulation → mechanical compression.
You’ll learn:
- Why castration causes rapid involution (proves this is hormone-maintained, not “aging scar tissue”)
- Why DHT is the real growth driver (testosterone → DHT via 5-alpha reductase)
- Why BPH isn’t “explosive cell growth” (mitosis isn’t wildly increased; cell death drops, and it compounds)
- The two-stage evolution:
- Early glandular hyperplasia (clean/symmetric)
- Later complex hyperplasia (stromal distortion + duct blockage + cystic change)
- The clinical paradox: the classic sign is often dyschezia/ribbon-like feces from rectal compression, while true urethral obstruction is less typical in simple BPH
Key takeaway: BPH becomes a disease when growth starts breaking mechanics.
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