The Migraine Treatment Guide Podcast
Medications, Procedures, and Surgery Explained for the management of chronic headaches, including migraine, tension headache, cluster headache, NDPH, and other headache diagnoses. Created and edited by Dr. Adam Lowenstein of the Migraine Surgery Specialty Center, this podcast covers diagnosis, medication, surgical, and non-surgical alternatives to headache medication in order to educate patients with chronic headache pain on their options for headache relief.
The Migraine Treatment Guide Podcast
Avoid Unnecessary C-spine Surgery
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A chronic headache that never lets up can make you feel like you’re doing everything right and still losing ground. We start with the nightmare scenario so many people live through: years of debilitating head and neck pain, endless treatments, clean scans, and then the crushing realization that the source of the problem may have been misread from the start.
We walk through the foundation of migraine and occipital neuralgia risk, from genetics that raise neuron excitability to anatomy that creates naturally tight “tunnels” for nerves passing through neck muscle and fascia. Then we connect the dots on why trauma matters so much. Whiplash and other neck injuries can trigger pain immediately, but they can also create a delayed mechanism where scar tissue thickens over time and slowly squeezes a peripheral nerve. That helps explain why a standard cervical spine MRI or CT can look normal while the patient feels anything but normal.
From there, we get into the most important distinction in the whole conversation: cervical nerve root compression at the spine versus peripheral occipital nerve compression downstream in soft tissue. Because the greater occipital nerve comes from C2 nerve root fibers, the brain can’t reliably tell where the pinch is happening. That overlap fuels a major diagnostic trap, including a common testing mistake where a cervical nerve root block can produce a false positive and steer someone toward invasive spine surgery like fusion even when the real issue is nerve entrapment in muscle. We lay out the safer sequence: test the periphery first with an occipital nerve block, then move upstream only if needed.
If you’ve been stuck in the chronic migraine, occipital neuralgia, or post-whiplash headache loop, share this with someone who needs a clearer roadmap and subscribe for more evidence-based breakdowns. After you listen, what question do you want to bring to your next neurology appointment?
For more information about nerve decompression for migraine headaches, occipital neuralgia, and other chronic headaches, call Dr. Lowenstein's clinic at 805-969-9004 and review HEADACHESURGERY.COM.
When Treatment Never Works
SPEAKER_02Um I want you to imagine a scenario that is, well, it's incredibly frustrating, but unfortunately, it's all too real for a lot of people.
SPEAKER_00Oh, absolutely. It's a very common nightmare.
SPEAKER_02Right. So imagine spending years, maybe even decades, living with this chronic, just debilitating head pain. And you are trying treatment after treatment. I mean, you're doing the physical therapy, you're taking all the prescribed medications, you know, you're changing your diet, you're getting multiple scans.
SPEAKER_00You're taking every box.
SPEAKER_02Exactly. And yet nothing is working. The pain is just always there, radiating up the back of your head, making normal life almost impossible.
SPEAKER_00Yeah, it takes over everything.
SPEAKER_02It really does. And then after all that time, you know, the money spent and the sheer suffering, you discover that you and your doctors have been looking for the source of the problem in completely the wrong place.
SPEAKER_00Aaron Powell Yeah, it's a devastating realization to come to because you know we tend to think of modern medicine as this perfectly precise map, right? Sure. Where every symptom leads clearly to a cure. But there are still uh there are still significant blind spots.
SPEAKER_01Yeah, definitely.
SPEAKER_00And for a very specific group of chronic headache sufferers, realizing they've been navigating one of those blind spots is an incredibly common experience.
SPEAKER_02And that is exactly our mission today. We are doing a deep dive into a really eye-opening medical document that uh, well, it breaks down the true causes of migraine and occipital neuralgia. We're gonna map out exactly how these severe headaches develop over time, and we are gonna uncover a major diagnostic trap in modern neurology.
SPEAKER_00A trap that catches a lot of very smart doctors, I might add.
SPEAKER_02Right. And most importantly, we're going to learn how distinguishing between two very similar sounding nerve issues can literally save patients from unnecessary invasive spinal surgeries.
SPEAKER_00Which is huge.
SPEAKER_02Absolutely huge. Okay, let's unpack this.
Genetics And A Tight Anatomy
SPEAKER_02Because to understand why things go so wrong in the treatment phase, we uh we first have to understand the baseline.
SPEAKER_00Where it all starts.
SPEAKER_02Yeah. Like if we're looking at two different people, why is one person just naturally more vulnerable to these massive headaches in the first place?
SPEAKER_00Aaron Ross Powell Well, we have to start at the foundation, which brings us to a mix of genetics and anatomy.
SPEAKER_01Okay.
SPEAKER_00Because there is a very well-documented hereditary component to migraines. I mean, a lot of patients can point straight to a parent or a sibling who has the exact same pattern of headaches.
SPEAKER_02Aaron Powell So it literally runs in the family.
SPEAKER_00Exactly. And researchers have actually identified specific genetic variants for this. Like, for example, there are mutations that affect calcium channel function in the brain.
SPEAKER_01Oh, wow.
SPEAKER_00Yeah. And those are linked to certain inherited migraine syndromes.
SPEAKER_02Yeah, some chests. So um we're talking about how the cells are chemically communicating with each other.
SPEAKER_00Right. It drastically changes the excitability of those neurons. You can basically think of it as the volume dial on the nerve being turned up too high.
SPEAKER_01Ah.
SPEAKER_00So the nerve fires off pain signals much, much more easily than it should. But for the vast majority of patients, there isn't just one single, you know, headache gene that you can isolate on a simple blood test.
SPEAKER_02Right.
SPEAKER_00The tendency toward getting migraines is usually polygenic.
SPEAKER_02Meaning a whole collection of different genes working together.
SPEAKER_00Exactly. It's this complex combination of many small genetic factors that constantly interact with a person's environment. It's a recipe, not a single ingredient, that determines who develops a migraine and just how severe that inflammatory response is going to be.
SPEAKER_02That makes a lot of sense.
SPEAKER_00But genetics is really only half of that baseline story, though.
SPEAKER_02Okay, what's the other half?
SPEAKER_00The other half is purely architectural. It is the physical anatomical blueprint of the patient.
SPEAKER_02Like how they are literally built on the inside.
SPEAKER_00Precisely. There is this growing recognition in the medical field that some people are simply constructed in a way that predisposes their peripheral nerves to getting compressed.
SPEAKER_02Really? Just the way they're built.
SPEAKER_00Yeah, we're talking about microscopic variations in anatomy, like the thickness of the muscles in the back of the neck.
SPEAKER_01Yeah.
SPEAKER_00Or the density of the fascia.
SPEAKER_01Fascia is that uh that tough connective tissue, right?
SPEAKER_00Exactly. The tissue wrapping around those muscles. And it's also about the exact winding path a nerve takes as it travels from the spine through that tissue and up to the scalp.
SPEAKER_02Oh, I see. So it's kind of like a house's plumbing blueprint.
SPEAKER_00That's a great way to look at it.
SPEAKER_02Like if you have the pipes, the nerves routed through incredibly tight wall spaces, which would be the muscles and fascia in this scenario. Right. It is going to take significantly less pressure to cause a blockage or a leak compared to a house that was built with, you know, wide open walls.
SPEAKER_00Exactly. If the walls are already tight, even a tiny shift causes a massive problem. What's fascinating here is that this anatomical variability perfectly explains something that frustrates so many patients and doctors alike.
SPEAKER_01What's that?
SPEAKER_00It explains why two people can have the exact same stress levels, the exact same lifestyle, maybe even sit at the exact same type of desk looking at the same monitors all day long.
SPEAKER_01Right, doing all the same things.
SPEAKER_00Yeah. And yet they have completely different headache experiences.
SPEAKER_01Oh, wow.
SPEAKER_00One person gets a mild, stiff neck, and the other is just down for three days in a dark room with an occipital neuralgia flare-up.
SPEAKER_02So it's not that they're doing anything wrong. They just have tighter walls, to use that analogy.
SPEAKER_00Yes. The person with a severe headache simply has less physical tolerance for inflammation or muscle tension because their nerves are already navigating a much tighter anatomical
Trauma And The Slow Scar Trap
SPEAKER_00tunnel.
SPEAKER_02Aaron Powell Okay. So if I have these naturally tight biological walls in my neck, I'm primed for a problem. But people aren't usually born with these massive chronic headaches. Something actually sets off the cascade, right? Like does someone just wake up one day and the nerve is suddenly trapped?
SPEAKER_00Aaron Ross Powell Usually no. I mean it requires an event to push that tight anatomy over the edge.
SPEAKER_02Aaron Powell Okay, like what kind of event?
SPEAKER_00Aaron Powell Well, in this context, that event is very often physical trauma. We're talking about a whiplash from car accidents, sports injuries, a bad fall, or you know, really any direct blow to the head or neck. Trauma is one of the most common starting points for chronic unyielding head pain. The issue is that trauma affects the nervous system in a few different ways, which makes post-traumatic headaches notoriously difficult to sort out.
SPEAKER_02Right, because it's not a broken bone where you can just point to a fracture on an X-ray and say, there's the pain.
SPEAKER_00Exactly. First, you have the direct injuries. The trauma can injure a cervical nerve root right at the spine itself, like if you herniate a disc or disrupt a joint during the impact of a crash.
SPEAKER_01Ouch. Yeah, that makes sense.
SPEAKER_00Aaron Powell Or it can injure a peripheral nerve directly by stretching it or bruising it as the neck violently snaps back and forth.
SPEAKER_01Right.
SPEAKER_00But there is a third mechanism, and this one acts as a hidden threat.
SPEAKER_02A hidden threat, like a delayed reaction.
SPEAKER_00Yes, the delayed muscle-mediated mechanism. When you suffer trauma to the neck, the body immediately tries to heal that damaged tissue.
SPEAKER_01Which is what it's supposed to do.
SPEAKER_00Right. But during that healing process, the surrounding muscle and fascia can scar. The tissue tightens up, and it can actually permanently thicken.
SPEAKER_01Oh.
SPEAKER_00And as that scar tissue forms, it gradually narrows the tunnel the peripheral nerve has to travel through. This doesn't happen overnight. Right. This structural thickening happens over months, sometimes even years, after the original injury felt like it had completely healed.
SPEAKER_02Wait, I need to push back on this a little bit.
SPEAKER_00Sure.
SPEAKER_02Because if someone is in a bad car crash, they usually get scanned right away. They go to the ER, they get an MRI or a CT scan of their neck.
SPEAKER_00They do, yes.
SPEAKER_02So if the muscle is scarring and essentially crushing a nerve, why wouldn't a doctor see this happening and just address it?
SPEAKER_00It's a great question. The problem is that standard cervical spine imaging, the MRIs and CT scans ordered in the ER or by a typical neurologist, they're designed to look at structural issues of the spine itself.
SPEAKER_02Oh, I get it.
SPEAKER_00They are calibrated to look at the bones, the discs, and the spinal cord. A slowly developing peripheral nerve entrapment hiding out in the soft tissue of the neck muscle.
SPEAKER_01It just doesn't show up.
SPEAKER_00Exactly. It simply doesn't show up on those scans in a definitive way.
SPEAKER_02So it's invisible to the specific test they are running.
SPEAKER_00Yes. The patient is sitting there in absolute agony, and the treating physician is looking at a completely clean scan of the cervical spine.
SPEAKER_02That is so frustrating.
SPEAKER_00It is. Both of them misunderstand the post-traumatic headache. The doctor might focus on the original impact, or they might look at some mild normal wear and tear on the vertebrae and blame that.
SPEAKER_02Completely missing the soft tissue issue.
SPEAKER_00Right. Completely missing the fact that the nerve is being suffocated by scar tissue an inch away in the muscle.
SPEAKER_02That is maddening. You're looking right at the area but with the wrong lens.
SPEAKER_00Exactly.
SPEAKER_02And trauma doesn't just cause scarring, right? According to the sources we're looking at, it can also fundamentally loosen the structures holding the head up, which uh creates a whole different
Craniocervical Instability And Guarding
SPEAKER_02set of problems.
SPEAKER_00Yes, this introduces a really crucial piece of the diagnostic puzzle: craniocervical instability.
SPEAKER_02Okay, what exactly is that?
SPEAKER_00So the craniocervical junction is the region where the skull meets the very top of the cervical spine. It relies on a very complex web of ligaments to stay stable.
SPEAKER_02Ligaments being the connective tissue holding bone to bone.
SPEAKER_00Precisely. And when those ligaments become lax or loose, it causes massive problems.
SPEAKER_02Where does that looseness come from?
SPEAKER_00This laxity can come from cumulative mechanical stress over a lifetime. Or it can come from that traumatic whiplash we just discussed.
SPEAKER_01Right.
SPEAKER_00Or it can even come from underlying connective tissue disorders, like Ailer's Danlow syndrome or EDS?
SPEAKER_02Oh, yeah, I've heard of EDS.
SPEAKER_00Yeah. So when those ligaments are too loose, the vertebrae at the top of the neck shifts slightly more than normal during everyday movement. The head is basically bobbing around on a loose hinge.
SPEAKER_02Okay, I want to make sure I'm synthesizing this correctly.
SPEAKER_00Go ahead.
SPEAKER_02Because the neck is suddenly unstable and bobbing around, does the body try to build its own natural cast by permanently clenching the neck muscles to hold the head steady?
SPEAKER_00Yes, exactly.
SPEAKER_02Which in turn accidentally crushes those peripheral nerves passing through those already tight tunnels we talked about.
SPEAKER_00That is exactly what happens. It's called chronic muscle guarding. The central nervous system senses the instability, it panics, and it orders those neck muscles to lock down to protect the delicate spinal cord.
SPEAKER_02Which makes sense from a survival standpoint.
SPEAKER_00Right. But by creating that rigid muscular cast, the body massively increases the tension in the exact muscles that the occipital and other terriferal nerves have to pass through.
SPEAKER_02So the body's own defense mechanism becomes the very thing causing the excruciating pain.
SPEAKER_00It's a tragic irony, really. And this creates a highly complex, mixed picture for a doctor.
SPEAKER_02Because there's so much going on.
SPEAKER_00Right. You have a patient with unstable vertebrae, which might be irritating the nerves at the spine, and you simultaneously have chronic muscle guarding, which is crushing the peripheral nerves out in the soft tissue. Right. It requires incredibly careful sequential evaluation to sort out how much of the pain is coming from the spine itself and how much is coming from the surrounding muscle.
Root Compression Versus Nerve Entrapment
SPEAKER_02I want to pause here because this complex web of pain part spinal, part muscular, it leads directly to what our sources call the most critical distinction in all of headache care.
SPEAKER_00It really is the dividing line.
SPEAKER_02Right. The difference between the spine and the peripheral muscle.
SPEAKER_00Yes. We have to clearly define the difference between a cervical nerve root compression and a peripheral occipital nerve compression.
SPEAKER_02They sound really similar.
SPEAKER_00They do. And they feel similar to the patient, but anatomically and surgically, they are worlds apart.
SPEAKER_02Walk me through the geography of a nerve root compression versus a peripheral compression.
SPEAKER_00Okay, so a cervical nerve root compression happens right at the spine. It's usually located at the neural foramen, which is the little opening between two vertebrae where the nerve exits the spinal cord.
SPEAKER_02Okay, so right at the center.
SPEAKER_00Right. This is typically caused by a bulging disc, bone spurs, or spinal arthritis. The pain usually follows a broader pattern, often radiating down into the arm or the shoulder, along with the neck.
SPEAKER_01Oh, I see.
SPEAKER_00To fix this kind of mechanical compression, you need major spine surgery, a foramenotomy, a dissectomy, or a spinal fusion.
SPEAKER_02You are literally operating on the bones of the neck.
SPEAKER_00Exactly. It's a major structural intervention. But peripheral nerve compression happens well outside the spine, downstream in the muscles, the fascia, and those soft tissue tunnels we talked about earlier.
SPEAKER_01Tight walls.
SPEAKER_00Right. The pain here is highly concentrated at the base of the skull, and it radiates up over the scalp, sometimes all the way to the forehead or behind the eyes.
SPEAKER_02Okay, so how do you fix that one?
SPEAKER_00To fix this, you absolutely do not touch the spine. You perform a peripheral nerve decompression, which is a surgery strictly in the soft tissue to release the tight muscle or scar tissue.
SPEAKER_02Here's where it gets really interesting, because you might be thinking, okay, one is at the spine, one is out in the muscle. Just ask the patient where it hurts and figure it out.
SPEAKER_00If only it were that easy.
SPEAKER_02Right. But the anatomical trap here is wild. The source explains that the greater occipital nerve, which is the peripheral nerve out in the muscle, is actually formed from the fibers of the C2 nerve root at the spine.
SPEAKER_00That's the kicker. They show the exact same origin point.
SPEAKER_02So because they share that origin, the pain signals they send back to the brain feel identical to the patient.
SPEAKER_01Yes.
SPEAKER_02A pinched C2 root at the spine feels exactly like a pinched occipital nerve out in the muscle. The brain literally cannot tell the difference.
SPEAKER_00You can't. And if we connect this to the bigger picture, we can see exactly why so many patients are funneled into the wrong treatments.
SPEAKER_02It makes total sense now.
SPEAKER_00Right. Both conditions cause head and neck pain radiating over the skull. Both can happen after a car accident. Both can even exist in the same patient at the exact same time.
SPEAKER_01Wow.
SPEAKER_00If a doctor relies solely on the patient pointing to the back of their head and then looks at an MRI showing some minor age-related wear on the C2 vertebrae.
SPEAKER_02They're going to assume the spine is the problem. Because the nerves are so intimately connected, standard diagnostic assumptions are actually lying to doctors.
SPEAKER_00Unfortunately, yes.
SPEAKER_02Let's
The Nerve Block False Positive
SPEAKER_02look at the misdiagnosis trap and something the documents call the flawed nerve block.
SPEAKER_00This is where things get really tragic. A substantial number of patients undergo major highly invasive cervical spine surgeries, literally fusing vertebrae together in their neck, specifically to cure their headaches.
SPEAKER_02Which is terrifying.
SPEAKER_00It is. They go through the risks of the surgery, they go through the grueling physical recovery, only to wake up months later with the exact same headache. Unchanged.
SPEAKER_02Because the imaging showed C2 issues, the doctor assumed the spine was the culprit and they operated on the spine.
SPEAKER_00Right.
SPEAKER_02But the real problem was further down the nerve, wrapped up in the muscle. Now, I know doctors do tests before operating, so how are they missing this?
SPEAKER_00Well, the standard test used to confirm spine surgery is a cervical nerve root block. Under an X-ray, a doctor injects numbing medicine directly onto the nerve root at the spine. Okay. If the patient's headache goes away temporarily, the doctor concludes that the spine is the definitive source of the problem, and they proceed with the operation.
SPEAKER_02But the document points out a massive mechanical flaw with this logic. Let me try this analogy for you.
SPEAKER_00Let's hear it.
SPEAKER_02It's like you are trying to figure out why a lamp in your living room is flickering. To test it, you go down to the basement and you turn off the main power breaker to the whole house. Right. The lamp goes out. So you assume the breaker box is broken and you spend thousands of dollars replacing the breaker box. But actually, the breaker box was fine. The power cord on the lamp in the living room was just frayed.
SPEAKER_00Exactly.
SPEAKER_02You interrupted the signal at the main source before it ever reached the true problem.
SPEAKER_00That is a perfect analogy.
SPEAKER_02Yeah.
SPEAKER_00The cervical nerve block at the spine is turning off that main breaker. It anesthetizes the nerve root before it branches out and travels into the muscle.
SPEAKER_01Oh man.
SPEAKER_00So the patient's pain goes away because the signal is blocked entirely from the root. That positive result does absolutely nothing to tell you where along the pathway the compression truly sits.
SPEAKER_02It just tells you the signal stopped.
SPEAKER_00Right. It could be at the root or it could be a frayed cord three inches away in the muscle. The block interrupts the shared signal regardless, generating a false positive for spine surgery.
SPEAKER_02This raises an important question. If the spinal nerve block produces false positives and essentially lies to the diagnostic process, how do doctors correctly test a patient? Right. Like how do we find the frayed cord without turning off the whole house?
SPEAKER_00It relies on basic anatomical logic, yet it is skipped with alarming frequency.
The Right Order Of Testing
SPEAKER_00The correct diagnostic sequence is that a physician must always perform an occipital nerve block first.
SPEAKER_02Meaning a block out in the periphery, not at the spine?
SPEAKER_00Yes. You inject the numbing medicine at the peripheral site, out in the neck muscle, far away from the spine itself. You target the specific downstream area where the soft tissue tunnel might be crushing the nerve.
SPEAKER_02So the logic here is completely inverted from the spine block.
SPEAKER_00Exactly.
SPEAKER_02If the peripheral block relieves the headache, then you know definitively that the problem is peripheral. The entrapment is in the muscle because you only numbed the end of the line.
SPEAKER_00Yes. If the pain goes away, you found the exact spot in the soft tissue. But if you do the peripheral block in the muscle and the patient still has their headache, then you know the true source of the pain must be further upstream.
SPEAKER_01It has to be at the spine.
SPEAKER_00Right. If numbing the downstream nerve doesn't stop the pain, the compression hasn't been addressed. That is when a doctor can confidently say, This is truly a spinal issue, let's evaluate for cervical spine surgery.
SPEAKER_01Wow.
SPEAKER_00Skipping this step and going straight to the spine block means the surgeon is essentially flying blind.
SPEAKER_02So what does this all mean? How does this change the reality for people suffering from these conditions?
SPEAKER_00That's a game changer.
SPEAKER_02Because this isn't just theory, right? This is actively changing how medicine is practiced by specialists who actually recognize this blind spot.
SPEAKER_00Exactly.
Decompression Instead Of Spine Fusion
SPEAKER_00It shifts the entire focus of treatment from incredibly invasive spine surgeries to highly targeted, less invasive peripheral surgeries.
SPEAKER_02The source material actually points to a very specific real-world example of this approach in action.
SPEAKER_00Oh, yeah.
SPEAKER_02It highlights Dr. Adam Lowenstein and the migraine surgery specialty center. They work specifically with migraine headache and occipital neuralgia patients, and their entire approach is built on this exact sequential diagnostic logic.
SPEAKER_00Which is how it should be done.
SPEAKER_02Right. They aren't just guessing based on a pain map or a blurry MRI.
SPEAKER_00They systematically rule out the spine by testing the peripheral nerves first. And by doing that, they're able to bypass unnecessary spine fusions for a huge number of patients.
SPEAKER_02That is incredible.
SPEAKER_00It really is. If the peripheral block is successful, they provide long-term relief through a much smaller outpatient nerve decompression surgery.
SPEAKER_02Oh, so it's not even an overnight hospital stay.
SPEAKER_00Usually not. They simply go into the soft tissue, release the tight muscle or scar tissue that's choking the nerve, and leave the spine completely untouched.
SPEAKER_02It's the difference between demolishing the foundation of your house to fix a plumbing leak versus just opening up a small patch of drywall where you know the pipe is actually pinched.
SPEAKER_00That's a great way to frame it.
SPEAKER_02The precision of the diagnosis completely dictates the scale of the solution.
SPEAKER_00And that precision is what has been missing for so long in chronic headache care. We have to stop assuming that because pain is felt in the head and neck, it must originate in the bones of the cervical spine.
SPEAKER_01Right.
SPEAKER_00The soft tissue, the muscles, the fascia. They are a dynamic, constantly changing environment, especially after trauma. They're just as capable of causing disabling neurological pain as a herniated disc.
SPEAKER_02I
Advocate For The Right Workup
SPEAKER_02want to speak directly to you listening for a second, because the ultimate takeaway from all this research, from understanding the genetics, the trauma starring, the unstable ligaments, and the shared nerve pathways. Right. The main takeaway is the absolute necessity of sequential diagnostic testing. You cannot rely on assumptions. You cannot rely purely on where the pain is located, and you cannot rely solely on a standard spinal MRI to tell the whole story. No, you really can't. If you or a loved one are ever sitting in a doctor's office and you are told that a major cervical spine surgery is the only option to cure your chronic headaches, well you now possess a vital piece of knowledge. You know to stop and ask about a peripheral occipital nerve block first. You know how to test the lamp cord before you let someone replace the breaker box.
SPEAKER_00That's right. Knowledge really is the best defense against misdiagnosis. Understanding the mechanics of your own anatomy gives you the vocabulary to advocate for yourself.
SPEAKER_02It really does.
SPEAKER_00And they'll leave you with this thought. If our own natural healing processes, like forming scar tissue to heal a whiplash injury, or permanently clenching neck muscles to stabilize loose ligaments, can inadvertently create a lifetime of chronic nerve pain simply by shifting a few millimeters of tissue. Right. It forces a much larger question. How many other mysterious chronic conditions in the human body, things we currently treat as incurable diseases, might simply be anatomical miscommunication?
SPEAKER_01Oh wow.
SPEAKER_00Just tiny mechanical entrapments waiting to be correctly mapped and decompressed, rather than permanent conditions we just have to live with.