When T3 Doesn’t Work — How to Dose Thyroid Medication Correctly (Part 2)

Beyond the Thyroid – Episode 46: When T3 Doesn’t Work — How to Dose Thyroid Medication Correctly (Part 2)

In Part 2 of this T3 deep-dive, Dr. Dana Gibbs explains why T3 thyroid medication often fails because of dosing strategy—not because T3 is the wrong medication.

Building on Episode 45, Dr. Gibbs walks through the most common mistakes clinicians and patients make when starting or adjusting T3-containing medications. She explains why how you dose matters just as much as what you prescribe, and why mismatched T4:T3 ratios, cortisol dysregulation, and insulin resistance can derail even the best treatment plans.

You’ll learn how proper dosing, timing, and lab interpretation can transform T3 from a medication patients fear into one that works reliably and safely. Dr. Gibbs also revisits the case of a patient named Barbara, showing how correcting dosing strategy led to real, sustained improvement.

This episode is essential listening for anyone who has tried T3 and felt worse—or wants to get it right the first time.

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Episode Highlights

00:00 – Introduction and Episode Overview
00:43 – Why T3 Problems Are Usually Dosing Problems
02:23 – How to Dose T3 Correctly
06:02 – Common Mistakes with T3 and T4 Ratios
21:09 – Cortisol, Insulin Resistance, and T3
25:21 – Case Study: Barbara’s Journey
25:50 – Steps to Optimize Thyroid Treatment
35:54 – Final Thoughts and Resources

With the right system, T3 can be one of the most effective tools in thyroid care.

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✉️ Email Dr. Gibbs at drgibbs@DanaGibbsMD.com or visit https://www.danagibbsmd.com/ for more information.


Thank you so much for listening! Tune in on the next episode.


The medical information provided in this episode is intended for informational purposes only and should not be construed as medical advice. Always consult a qualified healthcare provider regarding any medical questions or concerns.

Transcript

dana-gibbs-md--she-_2_01-23-2026_131810 0:03

Welcome to episode 46. Today we're discussing more about one of the most frustrating problems that I see in my patients. So they finally found a doctor who believed them about their hypothyroid symptoms, and they prescribed you a T3 containing medication, but then when you tried it, you felt worse. Maybe wilting with fatigue, faint, maybe shaky or anxious and irritable with palpitations or maybe all of that. So you think, okay, I tried T3 and it didn't work, so maybe I have and insert unprovable mystery diagnosis here. Well, I have good news for you. We started discussing this last week, but I just wanna reiterate the problem with T3 is rarely the medication itself. So on today's episode, I wanna dig further into what might have gone wrong and how to fix it. So let's get started with today's episode. But first, if this sounds like you and you're looking for one-on-one help with your thyroid, you can book a free introductory call with me today at danagibbsmd.com/call and I just added another state license. Our newest is now Colorado, so come see me.

1:31

You're listening to the Beyond the Thyroid podcast. I'm your host, Dr. Dana Gibbs. I'm an ENT surgeon and hormone specialist. For years, I struggled with my own unrecognized thyroid problems before and even after I was regularly performing thyroid surgeries. Then, one day, I learned something that turned my health around and opened my eyes to the limits of mainstream medicine in treating more subtle thyroid abnormalities. I spent the next 20 years fine tuning my hormone expertise in disorders like Hashimoto's disease, perimenopause, and stress related illness. Come join me as I share this new approach to hormones that empowers you to take control of your own thyroid and hormone imbalances. Let's dive in.

dana-gibbs-md--she-_4_01-23-2026_132102 2:24

Hello and welcome back to part two of this special series on why T3 frequently doesn't seem like it gets people the results they were hoping for. If you're new here, welcome. I am Dana Gibbs. I help people with Hashimoto's and normal TSH get their energy back. If you've been enjoying the episodes, I have a huge ask. Be sure to follow and please take one minute to review the podcast. It's so simple and it'll help more people like you find this vital information source. It's very, very simple. You tap the three dots at the bottom corner of the episode, then tap, go to show, and then scroll down to write a review. I read every one of these and I really appreciate your support. Alright, today we're continuing the discussion that has started in the previous episode about the problems people experience when they finally find a provider to start them on T3 containing medications. So many patients come to T3 containing medications after years of being told their thyroid labs look normal in mainstream medicine and the internet is really full of people who are passionate about these medications, particularly the desiccated thyroid products that have a combination of T4 and T3, or liothyronine, which is synthetic T3, and either of these can be the right answer for some people, but every week there is a new patient in my office who started on T3 containing medication and then felt worse. Such disappointment when they thought this new medication was gonna be the answer for them. Now, if you haven't listened to part one of this series yet, you might wanna check out that first, because those issues are kind of the prerequisite for getting a good result from T3. So we focused on all the system issues, the things that are happening around the medication that can sabotage your experience with T3 before you even get to dosing. But today we're gonna talk about the medication itself, the dosing strategy, and the interaction with. Thyroid hormone and cortisol that can make or break your experience with T3. So this episode discusses conceptual reasons patient may or may not respond differently to thyroid medicine. So any medication or lab decision should be made in collaboration with your personal physician. These pieces are especially important for clinicians because this is where conventional training is leaving you completely unsupported. So after that, I'm going to introduce the system that I use that consistently helps patients feel better with minimal side effects and without the short or longer term risks that many doctors worry about. So if you're a clinician listening, I wanna say again, this is not about blaming doctors for less than stellar outcomes using T3. The American Thyroid Association guidelines acknowledge that T3 is useful for certain patients, but they really give no guidance on who that might be or how to do it safely. There's no rule book to follow if you wanna gain experience with this valuable treatment. And it's not dead simple, like just check the TSH and adjust T4 until it's normal and it may not be for everybody, but it helps gives my patients their health back pretty consistently and time after time. So let's walk through the remaining four situations that make it look like T3 doesn't work. I apologize to you if you do not have a medical background. I'm gonna do my best to break this down and make it straightforward, but it's about to get a little deep in here. Alright. The first situation is when the T3 dose is actually too low. Now, most clinicians assume T3 only causes problems when the dose is too high. But a dose that's too low is actually one of the most common reasons people feel worse when they start T3. The reason has nothing to do with being"sensitive", and I'm using air quotes here to T3. It has to do with physiology and the way that what few guidelines there are instruct clinicians to start T3. Endocrinologists are trained to begin with extremely small doses of T3, while usually continuing the full dose of T4 for that patient. The guidelines simply say, start low, but they give no instruction on reducing T4 or how to balance the two. They say start low and then wait several weeks to test which is the common way that thyroid is prescribed. Start, and then you wait several weeks before you change the dose again. So what happens is that a patient receives a tiny amount of T3, the smallest you can get, maybe even only two and a half micrograms. And if a patient still has residual thyroid function is making their own T3, remember T3 being the active thyroid hormone in your body and in your tissues. So if you have some conversion of T3 on your own, then symptoms are there because that isn't enough, because that internal production or conversion isn't enough, but giving a very small amount of T3 can actually suppress the body's own conversion or production of T3 pretty quickly for a few reasons. First being that the TSH falls. The TSH, which is the pituitary signal for more thyroid hormone, that signal falls, and that happens long before the patient receives enough active T3 to replace what was lost originally. So in effect, the patient ends up with less usable T3 than they had before they started treatment. That's when you see an increase in fatigue, cold intolerance, depression, brain fog, constipation, and a sense of"hey, this is worse than it was before." This is not an intolerance, it's an incorrect strategy. It's a mismatch between the dose and the physiology. If the person was already on levothyroxine to start with and then they get a suppressing dose of T3 on top of a T4 dose, that may be a little more than their body can comfortably handle then that's a problem. And as a reminder, high doses of T4 actually lower your T3 levels. This is proven in studies. There is no clinical test that measures the actual tissue levels of T3. So we're dependent on the serum T3 to see what's going on. And this problem then is mostly invisible to providers who are relying solely on the TSH and the free T4, and then maybe they're checking free T3, but it's notoriously inaccurate at low levels and really, really timing dependent. So the mid dose lab testing and twice daily dosing schedule that I recommend to my clinic patients is pretty crucial to understanding when your T3 levels in your blood are sufficient. In addition to that, it's important that you substitute T3 for T4 in a stepwise fashion, maybe once a week, rather than just adding T3 on top of what already might be a high T4 dose. I also titrate the T3 dose up on a schedule that I explain in my more comprehensive lecture sets on this subject. And generally I make at least me three small upward steps and then check in before a hold and a retest. Then fine tuning. So a lot of people who start T3 and do well initially find that after a few weeks, the dose needs to go up again. In short, really low T3 doses often fail because they suppress internal production of T3 without providing quite enough replacement, especially if you're not reducing T4. So then the patient is left worse off, not because T3 is wrong for them, but because the strategy was not correct. The second situation that I wanna talk about is the opposite. It's if somebody is taking more T3 than their body can safely use, and the symptoms then are gonna look like hyperthyroidism. And if this gets bad enough, it can be quite dangerous. So this is not because T3 is inherently dangerous, it's not really any more inherently dangerous than T4 except for one thing. There is a point beyond which the dose is truly too high because too much thyroid hormone of any kind pushes your body systems too fast. Particularly your cardiovascular system is the big worry and the big problem. So, high dose T3 is something I see advocated on blogs and interwebs and by influencers and sometimes even in the bodybuilding world. But I also see it in patients who've been self-adjusting their dose, who come to me taking 50, 75, even a 100 micrograms or more of T3 in the form of over the counter natural desiccated thyroid products or sometimes without any T4 at all on board. And when somebody has a really low TSH indicating lidual or no residual thyroid stimulation, and they're taking a higher dose of T3, the body might lose the ability to buffer the excess T3. It becomes a pure stimulating effect. And if they're on no T4, there is no T4 that your body can convert into the reverse T3 to damp things down, to buffer things down. So the tissues are experiencing a really severe high T3 signal and it's constant, but it's also fluctuating because they're not necessarily taking their dose on a correct timing schedule either. So then that patient will physiologically look like somebody who has Graves disease, which is hyperthyroidism and the same level of danger. And there it is truly dangerous. When you start looking for true high T3 irritability is almost always the first symptom that I see in my clinic when we have gotten the dose a little bit too high. So somebody might say,"Hey, I feel more short fused or more reactive than I usually did." But hyperthyroid symptoms also include weight loss, palpitations, anxiety, tremors, heat intolerance, insomnia, wasting of muscle tissue, widen pulse pressure, meaning you're systolic and your diastolic blood pressure numbers get further apart, and eventually heart failure. So do I see this at the doses I use in my clinic? No. But do they occur at doses that are truly excessive? Yes. Once again, dose and lab timing is essential for getting this right. So T3 begins to wear off very quickly after it's taken within two to two and a half hours. It starts, goes way up, and then it starts to wear off. So if you're taking it once daily, it's already a bad deal because the fluctuation in blood levels is so huge from that two to two and a half hour mark, down to just before you take the next dose. If you then delay the next dose until after a morning lab set, a next morning lab set, you will see the lowest level, even lower than it ever is on a daily basis. So imagine this scenario. Jane is given 25 micrograms of T3, and that's a pretty high dose by the way, and she takes it at 6:00 AM. Her level goes up fast, way beyond. If you were to check her at 9:00 AM you would see it's way beyond the upper limit of the reference range for free T3, and then by 9:00 AM, it's headed back down. But she doesn't go to the lab until 9:00 the next day. And I'm waving my hands around because I don't have a graphic of this handy, but if I did have one, it would look like the first drop on the Texas Titan roller coaster in reverse. So the level continues to go steadily down until it's even lower than it was at the usual time she takes the meds the next morning. So her physician will see that lab and see that very low level and think, oh, I need to raise the dose when it might already have been too high. So dosing must be two or three times a day and half as much at a time due to the short half life of the T3 in the blood. So even then, if you're doing early morning labs, the free T3 blood test will not ever if it's correct, it'll not ever be at the high end of the reference range because that's the lowest it ever gets all day. So you really want it to be mid of the reference range if you're gonna do labs that way. And it's super, super important to take the labs at the same time of day every time. Then really pay attention to those symptoms. Symptoms shortly after the dose should always be given importance especially if it's a rapid heartbeat or an irritability feeling with consideration that the dose is too high. Alright. The third situation is when your T4 dose is too high. And this is where I think most of all of the T3 failures that I see in my clinic tend to originate because a lot of patients come to me already taking too much T4. It doesn't necessarily show because the free T4 reference range is really wide. And because most clinicians do not realize the levels as too high. Because they're all testing people 24 plus hours after they last took their dose, and that lab timing completely misses the very high early absorption level a patient gets when they take levothyroxine once a day. Then, if they're prescribed T3 on top of that high, once daily dosing, then those hyperthyroid symptoms will start even when the T3 dose is still relatively low. High T4 dosing causes several problems. The first is that T4 suppresses TSH which makes the clinician believe the patient is adequately treated when they're not necessarily producing or converting enough t3 and low TSH actually slows T3 production in the thyroid. The next is those transiently high T4 levels in the blood that you get with once daily levothyroxine actually block the signaling for T3 conversion in peripheral tissues by accelerating the breakdown of the deiodinase type two enzyme that makes T3. The third thing is that excess T4 increases reverse T3 production. So high reverse T3 indicates accelerated breakdown of T3 in your tissues because the same enzyme that breaks down T3 is also the one that produces. Reverse T3, so that same enzyme's doing both jobs, and so they're both happening at the same time. There are some older studies that suggested that high reverse T3 actually blocks the action of T3 at the receptor, though this has not been repeated or conclusively proven in recent years. I test the ratio of total T3 to reverse T3 as an indicator of T3 production versus T3 breakdown, and it's clear to me that when this ratio is low below nine, that people don't resolve their symptoms even if their T3 kind of looks high. So even if you're taking T3, your body is not getting the use of it, breaking it down very fast. If you add T3 on top of a high T4 environment, then you get those hyperthyroid symptoms. Again, the peaks feel like anxiety, jitteryness diarrhea, panic or heart racing. And then the clinician is assuming the patient can't tolerate T3. But this is once again, not generally a T3 problem. It's a T4 overdose problem. Here's one other thing that I learned recently that is new studies that are not human studies yet, but they're being done in mice, which is high T4 levels because of the high level of iodine, T4 being a molecule that has four atoms of iodine on it. That high T4 actually stimulates the cellular destruction of the thyroid gland in people who have autoimmune thyroid disease or Hashimoto's disease. So, this is yet another reason why too high of T4 is not a great thing. But anyway, if your TSH is below the reference range, something may be too high and almost always when I first see the patient that something is the T4 level, not the T3. So changing the dosing and testing schedule for T4 to every 12 hours and mid dose testing reveals that overdose lowering the T4 then allows reverse T3 production to fall and when the T3:RT3 ratio improves, gets above 10, the TSH then can rise back to an average TSH, which for me, I like to see it in the lower part of the reference range. So, when we divide T4 and reduce it into that more physiologic range, the reverse T3 can come down and normalize the peak smooth out a little bit, and T3 starts working much, much better. So this is actually the correction that changed Barbara's life. She's the lady I mentioned last week, and this is the most common fix that I find myself making in my practice. So, i'm gonna mention one more situation and that is a cortisol mismatch and it shows up in two different and kind of opposite ways. We also talked about insulin resistance last week, and this is important, so I'll just repeat that one too. So, T3 increases the speed at which your cells burn energy. So if insulin resistance is present, then you need more insulin to get the same response to a high glucose meal. So, in the presence of T3, once you burn through that glucose, the high insulin level is still there, and then you get this rollercoaster-like drop in your blood sugar, your brain senses the low blood sugar and ordinarily it'll respond with cortisol release from the liver. And if this doesn't happen fast enough because you're super stressed out and your cortisol release is maxed out, what happens is it sends out adrenaline instead. And that's when you get shakiness, irritability, anxiety pounding heart, sweating, panic like situations. And this is not the T3 itself, but an increase in the receptor sensitivity of the beta adrenergic receptors and those receptors are in your heart and your vascular system, so any adrenaline excess that you get is gonna feel even stronger. This problem of co-occurring insulin resistance really needs to be handled with improved dietary protein, lower quick release carbs, low sugar, correcting the intensity of your exercise and the quality of your sleep. And sometimes with medications before, some people will tolerate T3. The second one that is related to cortisol is the reaction of your body not having sufficient cortisol when you need it. So cortisol is required to maintain stable blood sugar, to support mitochondrial energy production, and to buffer the acceleration that T3 creates because it needs to raise your blood sugar. So when cortisol is too low, then your body can't meet that increased metabolic demand. And this happens for a variety of reasons. And most of them are situations that can trigger a chronic stress response in your body, which raises your need for cortisol. And the one that I'm gonna mention, we've talked about this in other episodes, but the one I'm gonna mention is excess. Calorie restrictions, calorie restrictions in excess of your basal metabolic rate. So if your cortisol production stays high, the nervous system becomes more sensitive and accelerates your sympathetic baseline tone. And if cortisol capacity gets overwhelmed, the body's gonna use adrenaline to compensate. But this person is gonna begin to look like what some providers call adrenal fatigue and the symptoms there are worsened, fatigue, weakness of your muscles, dizziness, nausea, low blood pressure, low pulse, craving salt, frequent urination at night, and even fainting. And it occasionally, it's as simple as discontinuing a supplement that a patient might be taking that blocks their cortisol response, their ACTH driven cortisol response. But it's almost never true Addison's disease. Addison's disease is truly a rare disease. So this cortisol mismatch is really the closest thing I ever see to true T3 intolerance, and it's almost always reversible once we stabilize the person's cortisol, their ability to produce cortisol, the rhythm of their cortisol. And this needs to be done through lifestyle changes and some supplements sometimes. And once that happens, the T3 becomes not just tolerable, but really transformative of people's energy levels. Alright, so now that we've walked through these situations, you can see how easily T3 can really look like it's the problem when in reality the situation requires more thoughtful care than just throwing on some T3. So, at the end of this, now I'm gonna go ahead and walk you through Barbara's case again, just to demonstrate how the system I use in my practice works to make T3 safe, predictable, and incredibly effective with very few side effects and no long-term complications. This is the system that has evolved and my practice over two decades of prescribing thyroid, and it's the system that consistently produces these good outcomes even in tough cases. So let's do step by step. So, step one is getting the right labs at the right time. This patient came in with a standard set of labs that already showed a fairly low TSH below the reference range, fairly high T4, still barely within the reference range. And what looked to me like a low-ish T3. It was still in the reference range, but it was on the low side. So, because I've been doing this for a while, and because I knew that these had been done the day after her last dose of T4 and T3, I already knew her T4 dose was higher than I wanted. And because I was gonna divide her thyroid dose already, her Synthroid dose, lower it a bit. She then waited a couple of weeks after that change to come in and get the new complete set of thyroid labs. And these were done on my recommended timing schedule. For most patients at this stage, I'm gonna also add labs, looking for those confounders that I mentioned last week, like low iron or elevated homocysteine, or new onset of perimenopause, depending on what the patient's history is telling me is the most important and likely problems for them. And depending on what labs they have already had done, because some people come back in or come in originally and they've had a lot of this stuff already done. They just haven't done anything about it yet. Alright, step two. We're waiting for the labs to come back. Now it can take up to two weeks to get a reverse T3 result back from the lab. So while I'm waiting for those labs to come back, I start doing a supplement evaluation, and then I start tweaking those supplements and gathering data on the patient's current eating habits and their plans and looking for red flags for insulin and cortisol problems. If I see those, then it may mean removing some supplements and adding others. The questionable ones we just take out, if it turns out the patient needs them later, we can always add them back in. I really try to make all these changes one at a time and wait three or four days in between changes. So I start that as soon as I possibly can with the patient. I have the patient provide a complete diet journal, and they're encouraged after they've done that for a few days, to start balancing the carbs that they eat with protein, removing the sugary snacks, stabilizing their timing of when they fall asleep or wake up supporting their sleep with minerals, correcting mineral and vitamin deficiencies, particularly D-3 if that's found. And you really shouldn't start T3 until you've got this foundation in place. The next step I'm gonna do is once the labs are back, the next step is re a really comprehensive review and informed decision making with the patient and we develop a plan. And that plan will be, we're gonna correct any other deficiencies that we find on the lab set, and then we decide where to go with the thyroid medication adjustments, or if we're gonna add T3, it is common to need to start reducing T4 before adding T3. And in Barbara's case, her new lab set came back with a T3 that looked much higher than it had on the prior day lab set. Actually, higher than ideal. So I could tell for sure she did not need more. T3, actually, I'd already guessed that because she was still having diarrhea every day. The TSH was extremely low, indicating that her total intake of T4 plus T3 was actually quite a bit higher than she needed and was responsible for several of her symptoms. And then the T4 was. Not outer range, but it was at the very top of the statistical reference range. Remember, this was a lab that hadn't been done mid dose with the divided T4 dose, so it was still at the very top, which means that at her lowest point, it's still quite high. So I was able to discuss with her what ideal ranges, she ought to have, and we decided on a gradual lowering of her T4 dose. So I discuss what those ideal ranges are and ought to be in my comprehensive courses for physicians and for patient oriented materials as well. So step four is picking a reasonable estimated target dose of T3, and I had two data points for her. I did not pick a massive dose and I didn't pick a tiny dose. Her case, I did not need to add any turned out she was actually already on a pretty good dose of T3, but I started lowering her T4, gradually in small steps until her hyperthyroid symptoms started to resolve. And her TSH started to rise back into the reference range. So when I start T3 for most patients, I do start them with the smallest possible dose given twice a day. But then the patient begins an escalation schedule that happens once a week to the correct amount that I estimated for her in the beginning. This prevents the low dose suppression problem we talked about earlier in this episode, it is important to split that T3 dose and to do our best, not only to split it, but to match their circadian rhythm. I rarely recommend a bedtime dose of T3 because it can be very stimulating. But remember, T4 is also getting divided for the reasons we talked about earlier, and we have to have a discussion about what other supplements and what foods can be taken or not taken along with thyroid hormones. These are not necessarily quite as strict as your pharmacist recommends, but there are some restrictions because if you, especially if you're varying what you eat and taking your thyroid meds along with food, then your absorption is gonna vary from day to day. That's one of the big reasons why taking your thyroid medication when your stomach is empty is a really good idea. And then waiting before you eat so that the absorption can happen before you put food in there on top of it. Alright, step five. We continue to draw the follow-up labs at the right time, and I order the total T3 and the reverse T3, free T3, free T4, TSH every time. It sounds expensive, but this whole set costs my patient less than$50, and that's their cash pay price. The T3:RT3 ratio is the single best indicator of whether the balance of T4 and T3 is. The correct one for that patient and the TSH then tells me overall they're taking too much or not enough hormone, and then the free hormone levels go up or down as needed. And occasionally the TSH will tell me something false. It'll remain low in somebody who's free t3 and free T4 levels are normal or even low normal. And so at that point then we say, you know what? We're gonna use the free hormone levels as our guide. And there are some other lab nuances that are mentioned in the physician course with regards to the type of lab test is used for these. There is more information about the type of labs to be used in my physician course. There are enough problems with thyroid lab interference and lab interpretation, actually for a whole other course module. So I do address that in my full signature physician course. For Just know that you really can't a hundred percent depend on them, and if they tell you something that doesn't make sense, it probably is wrong anyway. So step six then, is making fine adjustments to the thyroid levels while gradually watching symptoms, and once insulin and cortisol and minerals and circadian timing are aligned, T3 becomes actually easier to fine tune. I do retest after the initial escalation is complete after about two or three weeks after that. And then again, after any dose change or anytime the patient comes in and has had a change in symptoms, I also follow up pretty frequently and retest. Every three months for a whole year with no dosage changes. And this is where people are finally really beginning to feel their energy, returning their sleep normalizing, their digestion stabilizing their brain fog, clearing up their mood, smoothing out their vitality, coming back. This is when the patient's. Come in and look at me and say,"Hey, I had no idea. I could feel this good." So when people say, I tried T3 and it didn't work, what they usually mean is, no one ever showed me how to use T3 correctly, because T3 does work for people who need it. It works beautifully. And when the system around it supports its function, it works even better. So if you're a clinician listening, I hope this really gives you the resolve that you could do this for your patients. You know, they're in your office. We've been telling them for years that their symptoms are imaginary. They are not. You can absolutely use T3 in your practice and your patients will love it. Once you know how to avoid those pitfalls, and if you're a patient know this, you are not too sensitive, you are not intolerant, you've simply not been given the system your body needs to handle T3 correctly, and that is fixable. So if you'd like more help, whether you're a patient or a clinician, I have very soon upcoming the Thyroid Clarity Checkup which details the right labs to follow and how to understand exactly what they mean in your own context. You can find the information and the Priority List Page at danagibbsmd.com/checkup. All the links are in the show notes at danagibbsmd.com. So thank you for being here. You deserve to feel well, and you absolutely can. Until next time, I'm Dr. Gibbs.

36:37

Thank you for listening to this episode of Beyond the Thyroid. If you found this information valuable, it would mean so much to me to take a few seconds and give the podcast a five star review. It helps other people who need this information find the show and it's really easy. Just search and click on the name of the show, Beyond the Thyroid, and scroll to the bottom to ratings and reviews. I truly do read and appreciate. Remember, when it comes to hormones, there will always be more to discover, so follow the show so you get the next episode as soon as it's released. And if you or someone you care about needs a caring doctor to help figure out how to heal hormone problems that other doctors have dismissed, check out my website at www. danagibbsmd. com. And if you're not a physician, please keep in mind, while I'm a doctor, I'm not your doctor. The content of this podcast is my opinion and it's for educational and entertainment purposes only. This is not meant to be individual medical advice and you should consult your own physician for any medical issues or diagnoses that you may have. I look forward to continuing this journey with you beyond the thyroid.